| Acne is rarely serious,
but it often causes emotional distress and can lead to acne-caused
lesions of the skin. With the right acne treatment, you can often keep
breakouts under control. Measures can also be taken to reduce scars
left by blemishes. Untreated
or non-responsive skin breakouts (severe acne) may results in acne scar formation, a scenario more difficult to treat. Some figures of
acne never cause scar development, however, severe
forms are very likely to be followed by acne scarring. Also
rosacea in some individuals is more prone to occurrence of a scar,
which may suggest genetic factors as a predisposing factor. Mechanical
stimulation of lesions is another mode of causing damage ,at cellular
level, in sebaceous follicles in dermis level. Appropriate acne treatment
early and adequately is the best preventive method. Severe
acne can be treated using combination therapy, tretinoin and benzoyl
peroxide washes are one of the choices. Scar Pathology Injury to the skin
triggers wound healing mediations. Injury and consequent skin tissue
repair mechanisms cause the formation of new tissues, initial scarring.
Migration of inflammatory molecules at the site of injury (acne
also cause an inflammation and small injury at the sebaceous follicle
level, severe cases is usually associated with more inflammation)
and their repair will followed by a disordered site. This means
when the job of these molecules is done they leave a repair site
with an structure similar to a form of fibrous tissue, or eroded
tissue. Forming tissue is the sequel of improper deposition of collagen
and elastin and insufficiency of wound healing system. Epithelium
lining acne scars is not flat and atrophic but hyperplastic.
Healing them involves stimulation of the skin's healing process
and rebuilding collagen and elastin tissue. Hypertrophic scars are pink, raised,
firm, erythematous, characterized by a decreased expression
of collagenase (the enzyme that breaks collagen). Hypertrophic lesions
occur when collagen synthesis exceeds collagen lysis during the
remodeling phase of wound healing. The result is formation of thick
collagen bundles consisting of fibroblasts and fibrocytes. Despite
obvious tissue proliferation, these lesions remain within the confines
of the original integumental injury. Hypertrophic scars and their
keloid counterparts usually form in body areas that exhibit slow
wound healing or in pressure-dependent or movement-dependent areas.
Scarring tends to form within the first month following injury. Approximately
one third of patients report pruritus and dysesthesia. Unlike keloids,
hypertrophic skin damages may regress over time. Most scarring developed as a result of acne is ice pick scar. In particular, cystic acne, red tender nodules tend to leave this type of scarring. Ice pic scars are generally depressed lesions with uneven surface. In contrast, a keloid is raised, nodular,
more reddish scar that appear firmer than hypertrophic irregularities.
Keloids exhibit a prolonged proliferative phase, which results from
an inherited metabolic alteration in collagen. The result is thick
hyalinized collagen fibers. Unlike hypertrophic scar, keloids
extend beyond wound margins and do not regress over time. Keloids
also contain increased hyaluronidase. Formation may occur over weeks
or years following the initial trauma. Although they occur in all
skin types, keloids are most common in patients with darker skin.
Available modalities: There are no direct association between
types of acne lesions and likelihood of acne scar development. However,
cystic acne with inflammation tend to leave sequelae
more readily. At the beginnings acne scars present redder and thicker,
then gradually fade. In a few months they heal spontaneously without
any cure. It would be wise to wait sometime before considering
any treatment for acne scars. Most at home or office procedures for treating acne scars one way or another,
rely on skin renewal. Among removal methods laser resurfacing, dermabrasion
and chemical peels reach their ultimate goal through epidermal resurfacing
and renewal. Acne scar type and depth are two important factors
in determining the outcomes of these removal methods. Deep scars
as a result of acne not completely disappear using these
maneuvers. Available skin resurfacing modalities have improved
over the years. The older, less successful treatments such as dermabrasion
and chemical peeling have been replaced by the use of resurfacing
infrared lasers such as CO2 lasers13 and, more recently, erbium
lasers14 to better remove and tighten the skin. However, none of
these recent skin resurfacing modalities guarantee complete eradication of an acne scar. An understanding that replacement of
the diminished structures in the dermis and subcutaneous tissues
is necessary in severe cases has led to the development of superior
dermal and subcutaneous augmentation techniques. Dermal grafting, lipocytic dermal injectables, fat transfer and,
presently, the implantation of autologous collagen are examples of dermal and subcutaneous augmentation techniques. Foriegn tissue
augmentation is also possible by injectables such as of bovine collagen
and hyaluronic acid, among the most popular ones. Dermabrasion could be of help for acne scar treatment resulted from accidents or disease. This measure can be
performed in the dermatologic surgeon's office or in an outpatient
surgical facility. It is used to improve severe forms of wrinkling as well as other skin conditions such as a sun damaged skin. Anesthesia should be given prior to operation.
Cleansing and freezing of the skin is performed before using the
abrasive device. A high-speed rotary instrument with an abrasive
wheel or brush removes the upper layers of the skin and improves
irregularities in the skin surface. Tissue healing occurs within
one to two weeks after operation. Medications will be used to alleviate
the pain associated with the treated skin. Pink to red skin will
stay for as long as three to four weeks after dermabrasion. The
skin will be smoother as a consequence, total eradication should
not be expected. Superficial scars show more positive results. The type of laser used
is determined by the sequels that the laser aims to accomplish.
Lasers can vaporize skin mantle wise, causing slight cellular damage
to other tissue. Special scanning devices move the laser light across
the skin in predetermined patterns, ensuring proper exposure. Removal
of acne scarring, spots, lines around the eyes, mouth, and cheeks are the
primary uses for laser operations. Smile lines or those associated
with other facial muscles tend to reappear after laser resurfacing.
Post-cure redness stays for several months. Laser scar resurfacing appears
to achieve its best effects as a spot cure; patients expecting complete
elimination of their skin lesions will not be satisfied. Complete
removal is closely correlated with their prototypes. Peeling: This involves
the use of a chemical to remove epidermis, the most outermost layer
of the skin, in order to smooth depressed acne scar and give the surface
a more even color. It is most helpful for shallow superficial scar lesions.
Peels are divided into three classes: superficial, medium-depth,
and deep. The sort of peeling depends on the strength of the chemical
used, and on how deeply it penetrates. The first are used for fine
wrinkles, sun damage, rosacea, acne and extraneous healing. The
medium-level peeling is used for more obvious wrinkles and sun damage,
as well as for precancerous lesions like actinic keratoses and scar
tissue. Deep peels are used for the most severe forms of wrinkling,
scar revision and sun damaged skin. Go to next page.
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